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Researchers warn that this finding does not imply that cancer patients can take aspirin without a medical prescription.
In contrast, thromboxane A2 levels reduced in macrophages from hearts of newborn mice lacking Mertk and adult mice post injury, indicating an age and MerTK-dependent arachidonic acid metabolism in ...
As researchers explain in a release from the University of Cambridge, aspirin can reduce a clotting factor known as thromboxane A2 (TXA2).
Low-dose ("baby") aspirin irreversibly blocks the production by platelets of a substance called thromboxane A2. Thromboxane A2 both stimulates activation of new platelets and increases platelet ...
But in adults, this process did not work the same way—after an injury, their macrophages did not produce enough thromboxane A2, limiting their ability to regenerate heart tissue.
A team of researchers from the University of Cambridge believe they have uncovered the mechanism behind how aspirin, a common and inexpensive painkiller, might be able to stop cancer from ...
But in adults, this process did not work the same way - after an injury, their macrophages did not produce enough thromboxane A2, limiting their ability to regenerate heart tissue.
Not only did the action of macrophages trigger the production of thromboxane A2, unexpectedly stimulating heart muscle cells to multiply, ...